Monsters in my blood


In the Blood Stream,besides the red and white Blood Cells, there is a host of “small wiggily things.”This is so normal that those who analyze Blood call them “noise.                                                                                                                                            “These are small                                    Lifeforms:Bacteria,Viruses,Mycoplasmas,Parasites,Micro-filarial Worms,Molds, and Fungi all in different stages of their multi-stage development.

It has been found that Electromagnetic Energy created by a pulsating square wave can kill them as they pass through it.the Blood Electrifier was designed to do this, non invasivly. With it’s four cycle per second, direct current pulses,alternating positive and negative,applied to the wrist arteries for two
hours A day for 21 days, it gets at and cleans all the Blood in the Body.


Creepy Creatures attacking red Blood Cells in Vancouver
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    Ultimate Cancer Care



Please view these two educational videos to aid you in your decision making.

 South Korea, scientists used a magnetic field to get cancer cells to actually self-destruct. The body removes old, defective, and infected cells through the process of programmed cell death (PCD), or apoptosis. In apoptosis, the rejected cell responds to certain signals sent by the body by fragmenting. Immune cells then consume these fragments. The magnets help trigger apoptosis. When apoptosis fails, however, rejected cells divide uncontrollably, developing tumors.

Magnets Induce Apoptosis
Professor Jinwoo Cheon of Yonsei University in Seoul and a team of scientists conducted experiments on bowel cancer cells using magnetic fields to induce apoptosis. They attached iron nanoparticles to antibodies, which bind to “receptor” molecules on tumor cells. These molecules cluster when the magnetic field is applied, triggering the “self-destruct” signal and thereby apoptosis. In the experiment, over half of the bowel cancer cells were destroyed when the signal for apoptopic clustering came into effect. Untreated cells remained unaffected and unharmed.
The study is to be published in the journal Nature Materials.
Another surer way , if magnet therapy is not effective, is to go on baking soda therapy to balance body pH to 8, where cancer cells cannot live.  Cancer is the result of a state of imbalance… period!  Read” A CANCER THERAPY” BY Dr MAX GERSON.

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    Magnetic Pulser


The Magnetic Pulser      “Quotation” from  U.S. Patent 4665-898
You can’t get a Patent unless you can PROVE it works.


“The body part of an Person afflicted with malignant cells is disposed with a magnetic coil and subjected to a plurality of magnetic field pulses, the pulses having intensities of between about 1 and
about 100 Tesla and characteristic frequencies of between about 5 and about 1000 kHz. The pulsed magnetic field selectively inactivates and/or destroys malignant cells with relatively little damage to normal tissue as compared to conventional radiation therapy procedures. Herein, it is discovered that high intensity magnetic fields, applied in short pulses with moderate frequencies, can be used to selectively destroy or otherwise inactivatemalignant cells within tissue of a living animal. Selective inactivation of malignant cells within animal tissue subjected to a pulsed magnetic field is accomplished without noticeable deterioration of gross characteristics of normal tissue. Substantially no heat is generated in the tissues, even in tissue which is sequentially subjected to a high number of pulses.DETAILED DESCRIPTION OF A PREFERRED EMBODIMENTIn accordance with the present invention, animals or animal body parts are subjected to high intensity, moderate frequency magnetic field pulses to selectively kill or inactivate malignant cells within the tissues. The whole or portions of an animal body (the term “animal” is used herein to include humans, although initial experiments have been carried out on lower animals) can be subjected to the pulsed magnetic field. This process is carried out with a minimum effect on normal cells and without altering the gross characteristics of the subjected normal tissues.

It is found that subjecting body parts containing cancerous tissue to a plurality of magnetic field pulses, with characteristic frequencies above about 5 kHz and intensities above about 1 Tesla, will either arrest the growth of tumors or progressively reduce the number of cancerous cells, resulting in remission of tumors. Tissue treated with pulsed magnetic fields according to the present invention are not significantly heated, and thus there is no
thermal discomfort to the subject and no burning of tissue whatsoever. Unlike X-ray or other ionizing radiation techniques, inactivation of cells is not achieved by an ionization mechanism, and there is no apparent alteration of the gross and functional characteristics of normal tissue. The method is applicable to practically any type of tissue and is believed applicable for treatment of most types of malignancies. Furthermore,treatment with a pulsed magnetic field does far less damage to the natural immune system than does radiation treatment or chemotherapy. Frequently, a patient who is treated extensively with ionizing radiation and/or with chemotherapy will experience an almost complete breakdown of the immune system. Subsequent to treatment, the immune system may take up to a year to recover, particularly with respect to immunity to viral infections. As a result, even if a patient is cured of the malignancy by radiation and/or chemotherapy, he is subject to debilitating disease or even death by infections to which his body would ordinarily have built up immunity. with the magnetic treatment described herein, there has been no evidence of major immune system break-down.
Although malignant cell inactivation is effected in the absence of more conventional selective tumor cell destruction procedures, such as irradiation therapy or chemotherapy, it is understood that the magnetic therapy practiced in accordance with the present invention may be used in conjunction with other therapeutic procedures. ” End of Patent Quote. U.S. Patent 4665-898



healthy food to boost your immune system


How Sugar Intake can Impact Cancer Treatment Success Cancer loves sugar. Cancer cells have about 12-38 times the receptor sights for insulin than normal cells. Insulin is the key receptor
that allows cells to use sugar as source of energy.Armed with the knowledge that cancer cells prefer glucose, we developed several proprietary cancer treatments to kill cancer cells and protect the
other “healthy” cells in the body. Discussions about a person’s diet go far beyond what we choose to eat, rather diets can be leveraged to assist patients win their fight against cancer.What Role does Diet Play in the Health of a Cancer Patient? The number one goal of a diet for cancer is to improve your immune system.The immune system is virtually ignored by the majority of cancer centers and hospitals. This area is called immunotherapy.
How does what you eat, or more importantly what you don’t eat, impact the immune system? It has to do with reducing toxic exposure to substances that occupy your immune system and detoxification pathways.Healthy Living in a Nutshell It is optimal to only consume organic whole foods such as fruits, vegetables, and grass-fed meats free of antibiotics and hormones. In our book The Stupid Diet, we strongly urge people to eat powerful, smart-impact foods with a holistic mind, body, and soul approach to eating. This allows us to achieve a healthy body weight and to have the best possible chance of avoiding illness through fortifying the body’s immune system. A normal body weight is an important goal to have when striving to live a healthy life. However, being thin isn’t always a perfect health indicator. Envita Medical Center CEO Dr. Dino Prato, author of The Stupid Diet, states, “Skinny people get cancer and degenerative disease too.”
Have you read all the recent studies suggesting that eating meat is at the root of all cancer? Well, there are two major flaws with that line of thinking.First of all, are those people eating enough vegetables and fruits as their diets’ main day-to-day staples? When people become vegetarian due to health or personal reasons, they tend to eat more fruit and vegetables and ultimately feel better.Second, for many centuries people have been consuming meats and still cancer rates were much lower than they are today. Quality fish and meats contain far more nutrients that serve to protect against cancers than the non-natural corn-fed meats sold at major grocery stores. Grass-fed meat contains more conjugated linoleic acids that actually protect against colon cancer.
It is definitely acceptable to choose a vegan or vegetarian diet if that is a lifestyle you can continually maintain. However, our clinical experience suggests that most cancer patients genuinely feel better and much more energetic with a diet containing high-quality meats. That being said, everyone’s metabolism is different and you need to find what works best for you. The most important aspect of refining your diet, is to improve the quality of foods you choose to eat.Eating Healthy in America: the Bottom Line Consider that roughly 67% of America is obese and that one in every two individuals, on average,will develop cancer or heart disease in their lifetime. It is clear that we must urgently change the way we take care of ourselves.Instead of following new diet trends and gimmicks, eat well and exercise as much as possible. Fad diet’s will ultimately detract from your main goal of health and can  increase the amount of weight you’ll put back on after going off of the diet.
The truth is, the dieting world is a crazy one. Invest in a good quality multivitamin and pick up a copy of the Stupid Diet (sold in all Envita offices). The time to learn how to get yourself (and your loved ones) on the right path, has come.

Crytomyces Pleomorpha, a Cancer causing fungus investigated by rife in 1935. I’m running it down now. It is now listed as a “new” organism.
At one stage of its pleomorphism it mimics the human blood cell. This may be the “perfect”human red blood cell that is found dessicated in chemtrails.


Tissue stained to reveal micro organisms,Vancouver
Myalgic Encephalitis, now more commonly called Chronic Fatigue Syndrome or Fibromyalgia. large fungal forms are present at the end-stages of cancer and Aids. It is recognised that frequently the
cause of death is due to systemic fungus infestations or mycoses. Conventional theory assumes that these are secondary to tumours or the AIDS virus, while the observed pleomorphic life cycle shows
that these and their fungal stages are the primary cause why people die of cancer and probably AIDS.The reason for the lethal effects of severe mycoses is probably a combination of poisoning of the
energy-producing mitochondria inside cells by fungal toxins and the destruction of erythrocytes by pleomorphics.  These pleomorphics not only fill the inside of red blood cells and deplete them of nutrients, they also form spines and long protrusions in the cell wall when they move out into the plasma. Someone with myasthenia gravis, an autoimmune disease, once mentioned that he was shocked to see that most of his red blood cells looked like black sea-urchins. These erythrocytes can no longer supply nutrients to the body and are quickly destroyed in the spleen.This is the real cause of severe anaemia that is so common in advanced cancer and various other diseases. In the end stages of cancer nearly 100% of erythrocytes are strongly infested and dysfunctional. This then leads to cachexia (muscle wasting with extreme fatigue) as the leading cause of death in cancer and AIDS. However, as shown in the longer video, with special Enderlein vaccines even in advanced cancer the erythrocytes could be returned to health within one month with simultaneous shrinking of existing metastases.
You may wonder how it is possible for a single cause such as an overgrowth of the blood with pleomorphics to lead to many different diseases. The answer is basically the same as why a cyclone or hurricane can destroy one building and leave another one undamaged, or rips off the roof of one and causes water damage in another. When the immune system is severely weakened then any pathogens have free range, and the weakest organ will be the first to crumble.


Dr. O. Cameron. Gruner Dr. Rife And Dr Gruners Cryptomyces Pleomorpha Fungus
On the Marsh Rife Audio CD’s Dr. Rife talks at great length about his work with Dr. Guner on the Cryptomyces Pleomorpha Fungus. Below is Dr. Gurners written paper on this organism.CRYPTOMYCES PLEOMORPHA: A NEW ORGANISM ISOLATED FROM THE BLOOD OF A CASE OF METASTASIZED CARCINOMA OF THE BREAST*By: O. C. GRUNER, M.D.,

Montreal The Canadian Medical Association Journal, Jan, 1935

THIS report is presented for the following reasons:

(1) The organism was detected in the circulating blood by direct examination (Fig. 1).

(2) It was detected among the tumour cells in the original neoplasm, in sections which had been made four years previously (Fig. 2).

(3) An organism, evidently of the same type, has been found in seven previous cases, but its cultural characteristics and pathology had not presented the same degree of completeness.

(4) Though in part resembling other fungoid organisms which have been described before in human pathology, this one seems to present additional distinctive and interesting features of its own.

(5) Careful observations of the living cultures have shown that in some phases this organism exactly mimics the cell-elements of human blood, and  the so-called Plimmer bodies and Russell fuchsinophile bodies of some malignant tissues, and, in addition, has forms which are just like the free nuclei and various sized granules found in many sections of neoplasms. These mimicries explain how the organism may effectually escape detection
in the routine observation of tissues and blood.

Isolation.-From the blood of a case of carcinoma of the breast (excised five years ago) with intrathoracic metastasis; previously, in other cases, from the tumour tissue.

Cultural characteristics.-The initial growth appeared only after seven days’ incubation at room temperature. After developing a raised formation like that of sporotrichum, but extremely hard in consistence, it remained stationary for two weeks, despite repeated attempts at subculture on a great variety of media of all gradations of pH. Finally, the use of the patient’s serum, and asparagus extract to enrich the glycerine-glucose-peptone
water, proved to induce cultural success, good growths now appearing in all subsequent cultures in 48 hours. The organism was strictly aerobic.Room temperature was best. The optimum pH was 6.8 to 7.0. Bacterial contamination effectually antagonized the cultures.

On a.sparagus agar.-Within 48 hours a small round orange-tinted colony appears with elevated glistening centre. As this gradually enlarges it becomes coral pink. A somewhat hardened pellicle forms in the centre and delicate threads form at the periphery, at first whitish, and, later, faintly coral-pink (Fig. 1).

Petri dish culture, 12 days old. The central raised growth consists of the torula-like spore forms, and is of coral colour in the original. The delicate hairy outgrowth is the mycelial phase. Only spore forms occur in the initial colony. Hyphae develop in from 6 to 10 days, showing various forms as the culture ages . After 14
days the pink centre wrinkles up, much like sporotrichum, Mycobact. tuberculosis, etc., and the whole growth constitutes quite a firm membrane rather firmly adherent to the surface of the medium. Finally, a very fine white powdery deposit appears all over the growth, and no further growth occurs. (SEE ABOVE PIC)

On asparagus glucose glycerine peptone water, a pellicle forms in 48 hours; this is in the form of a ring at the surface, adhering to the glass, and made up almost entirely of mycelia, the spores falling to the bottom of the tube in large numbers. A pink colour appears in 7 to 10 days, and the ring can be detached from the glass, when it will remain intact and hanging in the medium.
Gelatine.-Not liquefied; very slow growth.
Meat-extract media.-Very slow growth; colonies very small; whitish; no colour.

Plain glucose glycerine agar.-Extremelyslow growth; small glistening colonies; no colour; no mycelium.

Sabouraud.-Very slow colourless growth; very small and few hyphre; spores much smaller than those on asparagus media.

Dextrose-tartaric acid.-No growth (in distinction from usual fungi).

Potato.-Very scanty dull yellow growth, slowly turning pinkish.

Tomato.-Very luxuriant growth in 48 hours; pink colour slightly paler than the flesh of the fruit. The condensation water rapidly fills with torula forms and turns pink.

Milk.-Budding torula forms develop in 72 hours; no coagulation; no acid.

Peptone water.-Noindol.

Nitrate media.-No nitrite formation.

Sugar-reactions.-No fermentation of glucose; gas, but no acid in lactose; saccharose, nil.

Stainability.-Dilutecarbol-fuchsin gives the best results. Methylene blue stains relatively feebly. It is Gram-positive. It is not acid-fast. Rossophilia is almost absent.

Morphology.-This organism is very pleomorphic. Thus at different times, in the same culture tube, one may see transient micrococci like Doyen’s (1

) M. neoformans of 1904 (Fig. 8), spherical forms like Sanfelice’s blastomyces (1896) (Fig. 7), tubular forms apparently answering v. Brehmer’s (2) description of his Siphonospora (Fig. 8) ; oval resting spores like those found in a specimen of Schmidt’s (3) “Blastolysin” (Fig. 9); combined with mycelial formations (Fig. 11).
The following forms of reproduction are observable, as met with in other fungi: (a) fission forms or oidia, or diplospores (Fig. 10) suggestive of schiwsaccharomyces; (b) bud-formation as usual in torula or saccharomyces (Fig. 11); (c) ascospores, or “megaspores” (Fig. 12) in keeping with zygosaccharomyces; (d) very minute microspores (Fig. 14), which are highly refractile and arise by detachment from (b); these are metachromatic
with Leishman and with methylene blue; (e) chlamydospore formation (Fig. 15); (f) lateral buds (Fig. 28), as in typical sporotrichum; (g) sessile sporangia in the aerial mat (Fig. 16), suggestive of some mucors; (h) still smaller particles occur, possibly filter-passing. There is a fairly regularsequence. Thus from b to d, from b to mycelium (first week), from mycelium to f, and to a, and to e (second week); from a to c and to g. The whole series ends in about three weeks, when a new subculture becomes essential. The organism is therefore an ascomycete, having affinities with sporotrichum, cryptococcus and blastomyces. It is given the name of Cryptomyces because it combines mycelial formation with an obscure biology and incidentally an effective concealment when situated in invaded tissues.Pathogenicity. – It is pathogenic for white rats ,when given intrapleurally, and for white mice when given into the submammary tissue. It was recovered true to its original cultural characteristics in pure form from the blood and all organs of these animals but developed very little mycelium.
A high monocytosis was striking and early. The progeny of a female rat inoculated with the organism while pregnant developed generalized infectionwith the same organism, dying 21 days after birth. Subcutaneous inoculation produced indurated areas in which the organisms were plentiful. After 12 days nodules formed in which spindle-shaped connective-tissue cells were predominant, and megaspores were abundant among them.
Mycelium was not detected in the tissues.Cuti-reaction.-Skinreactions have been obtained by using an emulsion of this organism on the patient whence it was derived, as well as on several other malignant cases, including sarcomas, but not on persons in ordinary health. Further studies on this point are in progress.
Relation to malignant disease.-Naturally, such an organism may be primary or secondary. If the former it would presumably act in virtue of some carcinogenic substance being formed among the products of its metabolism. In any case, the frequency of its association with new-growths remains to be established. The usual arguments against a microbic origin of the disease naturally arise.


An accidental admixture of two or more organisms would provide a simple explanation of the pleomorphism. The two main forms have already been separately described from time to time as being causal for cancer, to be subsequently rejected as “contaminants”, namely, the yeast-like forms and the mucor-like forms. Moreover, in the present case, the cultures after passage through the infected rats and mice showed a dominantly blastomycetic form, with mycelial formation almost negligible. Further, the patient had been for a few weeks in a region where sporotrichosis occurs,though never having the slightest evidence of lesions of that kind.The reasons for believing the organism to be single arc: (a) the constancy of the occurrence of the dual forms in succession, at the same time intervals in all subcultures; (b) the ability actually to observe the one form changing into the other; ( c) the inability to separate them permanently by plating; (d) the cultural characters; (e) the possession of pathogenicity; (f) its unique character; (g) the unlikelihood of a chance admixture of distinct organisms resulting in such a close symbiosis as actually to manifest conjugation.

Of some interest is the experience of the animal lesions produced, in that the organism is found closely mingled with the reactive infiltration of  predominantly monocytic type, and shows in the sections appearances which strongly recall those seen in sections of carcinomas and sarcomas, though such particles are usually regarded as unessential bacterial or degenerative components. Applied to human tumour histology, the presence of such microscopic objects would ‘seem to require an explanation.
Of further interest is the evidence of sexuality in this organism (in common with other fungi), though the question of haploid and diploid phases has not been taken up. The delay in obtaining good subcultures would be explained by the difficulty of finding a medium favourable for the conjugation of the + and – elements. This provides a useful suggestion that such occupants of tumour tissue (whatever their presence means) present a sexuality factor which determines ease or difficulty of culture outside the body. The repeated observation of the megaspore forms inside the phagocytic cells in the infected animals with their mimicry of the Plimmer-body and other inclusions in human cancers supports this suggestionquite definitely.


A report is given of a new organism, here called Cryptomyces pleomorpha, which is placed among the ascomycetes. The question of etiological relationship with malignant disease is necessarily left open.
Very grateful thanks are due to Dr. Archibald and to Dr. Mark Kaufmann for enabling this case to be worked out, and also to Dr. Pauline Beregoff, for recently undertaking further tests on animals.


1. DOYEN: Ewing’s Neoplastic Diseases, W. B. Saunders, Phila., 3rd ed., 1928, p. 122.

2. V. BREHMER, W.: Krebs eine Erregerkrankheit, Fortschrit. der Medizin, 1932, 50: 697; edit. in Brit. M. J., 1934, 2: 520; annotation in The Lancet, 1934, 2: 609.

3. SCHMIDT, O.: Ewing’s Neoplastic Diseases, W. B. Saunders, Phila., 3rd ed., 1928, p. 125.

* From the Department of Surgery, McGill University, Montreal. Under a grant for Cancer Research contributed to Dr. E. ‘V. Archibald by an anonymous donor.

  Several species of mycoplasma are frequently detected in different types of cancer cells. These species are:

M. fermentans

M. genitalium

M. hyorhinis

M. penetrans

The majority of these mycoplasma have shown a strong correlation to malignant transformation in mammalian cells in vitro.(livin tissue)

Mycoplasma infection and host cell transformation The presence of mycoplasma was first reported in samples of cancer tissue in the 1960s. Since then there have been several studies trying to find and prove the connection between mycoplasma and cancer, as well as how the bacterium might be involved in the formation of cancer.  Several studies have shown that cells that are chronically infected with the bacteria go through a multistep transformation. The changes caused by chronic mycoplasmal infections occur gradually and are both morphological and genetic. The first visual sign of infection is when the cells gradually shift from their normal form to sickle shaped. They also become hyperchromatic due to an increase of DNA in the nucleus of the cells. In later stages, the cells lose the need for a solid support in order to grow and proliferate as well as the normal contact dependent inhibition.

Possible intracellular mechanisms of mycoplasmal malignant transformation Karyotypic changes related to mycoplasma infections

Cells infected with mycoplasma for an extended period of time show significant chromosomal abnormalities. These include the addition of chromosomes, the loss of entire chromosomes, partial loss of chromosomes and chromosomal translocation. All of these genetic abnormalities may contribute to the process of malignant transformation. Chromosomal translocation and extra chromosomes help create abnormally high activity of certain proto-oncogenes. Proto-oncogenes with increased activity caused by these genetic abnormalities include those encoding c-myc, HRAS, and vav.  The activity of proto-oncogenes is not the only cellular function that is affected; tumour suppressor genes are affected by the chromosomal changes induced by mycoplasma as well. Partial or complete loss of chromosomes causes the loss of important genes involved in the regulation of cell proliferation.  Two genes whose activities are markedly decreased during chronic infections with mycoplasma are the Rband the p53 tumour suppressor genes. A major feature that differentiates mycoplasmas from other carcinogenic pathogens is that the mycoplasmas do not cause the cellular changes by insertion of their own genetic material into the host cell. The exact mechanism by which the bacterium causes the changes is not yet known.

Partial reversibility of malignant transformations The malignant transformation induced by mycoplasma is also different from that caused by other pathogens in that the process is reversible. The state of reversal is, however, only possible up to a certain point during the infection. The window of time that reversibility is possible varies greatly; it depends primarily on the mycoplasma involved. In the case of M. fermentans, the transformation is reversible up until around week 11 of infection and starts to become irreversible between week 11 and 18. [21] If the bacteria are killed using antibiotics  (i.e. ciprofloxacin  or Clarithromycin  before the irreversible stage, the infected cells should return to normal.

Connections to cancer in vivo and future research Though mycoplasmas are confirmed to be carcinogenic in vitro, it is not yet confirmed whether mycoplasma might be an actual cause of cancer in vivo. The uncertainties regarding the bacteria’s potential to cause malignancies is mostly due to the fact that the cells used for the studies are most often from immortalised cell lines like the BEAS-2B cells. These are essentially cells on the verge of becoming cancer cells. One big problem with using these cells to confirm carcinogenic properties is that they will transform spontaneously after 32 passagings (when a small number of cells are transferred into a new vessel to extend culture duration).  This, and the fact that no malignant transformation has been detected in non-immortalised “normal” cells that have been infected, might be an indication that mycoplasmas accelerates a cell’s progression towards malignancy, rather than actually causing it. No mycoplasma-generated cancer has yet to be documented in in vivo cultures. It might, however, be possible that very long, chronic infections of mycoplasma are able to cause cancer in non-immortalised cells. This is not yet known since non-immortalised cells can only divide for a limited number of times, and therefore it has not been possible to keep culturing them long enough to develop cancer. More research is needed to confirm that mycoplasma infections cause cancer or initiate malignancies in human cells. This might be an important step to treat and prevent cancer.

Types of cancer associated with mycoplasma Colon cancer: In a study to understand the effects of mycoplasma contamination on the quality of cultured human colon cancer cells, it was found that there is a positive correlation between the amount of M. hyorhinis present in the sample and the percentage of CD133 positive cells (a glycoprotein with an unknown function). Further tests and analysis are required to determine the exact reason for this phenomenon.

Gastric cancer: There are strong indications that the infection of M. hyorhinis contributes to the development of cancer within the stomach and increases the likelihood of malignant cancer cell development.

Lung cancer: Studies on lung cancer have supported the belief that there is more than a coincidental positive correlation between the appearance of Mycoplasma strains in patients and the infection with tumorigenesis. Because this is a such a new area of research, more studies must be performed to further understand the correlation.

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